Stage 6: Anxiolytics & Sedative-Hypnotics
Concept 3 of 10
R6.3

Lorazepam (Ativan)

Intermediate-acting BZD — IV available, no active metabolites, workhorse for acute psychiatric situations.

Lorazepam versatility: PO, SL, IM, IV — covers every acute situation. Multiple settings: ED agitation, alcohol withdrawal, status epilepticus, catatonia, procedural anxiolysis.

Lorazepam — Ativan — is the workhorse benzodiazepine for acute psychiatric situations. It is the most versatile BZD in routine practice: available oral, sublingual, intramuscular, and intravenous; with predictable kinetics; with no active metabolites that complicate hepatic impairment; and with a half-life around 12-18 hours that balances onset against accumulation.

Drug card
Class
Intermediate-acting benzodiazepine
Mechanism
GABA-A positive allosteric modulator
Typical dose
0.5-2 mg every 6-8 hours; IV/IM 1-2 mg for acute agitation, status epilepticus, alcohol withdrawal
Half-life
~12-18 hours
FDA indications
Anxiety, acute agitation, alcohol withdrawal, status epilepticus, catatonia, procedural anxiolysis
Key adverse effects
Sedation, cognitive impairment, ataxia, respiratory depression at high doses or with opioids, tolerance, dependence

Black box: Concomitant opioid use; abuse potential and dependence

Multiple formulations (PO, SL, IM, IV) make it versatile. No active metabolites — useful in hepatic impairment (vs. diazepam). IM/IV for acute agitation often combined with haloperidol. First-line for catatonia (often dramatically effective). Alcohol withdrawal: lorazepam (or chlordiazepoxide for longer-acting).

For acute agitation in the ED or inpatient unit, IM lorazepam is the standard adjunct to antipsychotics. The classic combination — IM haloperidol 5 mg plus IM lorazepam 2 mg — produces calming effect within 30 minutes with mechanism synergy.

For alcohol withdrawal, lorazepam (or chlordiazepoxide for less severe outpatient cases) is first-line. The Clinical Institute Withdrawal Assessment (CIWA) protocol uses lorazepam doses titrated to symptoms; severe withdrawal can require very high doses. In hepatic impairment from cirrhosis, lorazepam's glucuronidation-only metabolism makes it the preferred BZD — diazepam and chlordiazepoxide accumulate dangerously in liver disease, but lorazepam, oxazepam, and temazepam don't.

Mechanism in practice

Lorazepam is an intermediate-acting benzodiazepine whose simple, extra-hepatic metabolism makes it a workhorse in medically complex patients.

Mechanism
GABA-A positive allosteric modulation
Effect
Anxiolytic, sedative, anticonvulsant effect
Clinical applications
Used for acute anxiety, agitation, catatonia (diagnostic and therapeutic), status epilepticus, and alcohol withdrawal.
Mechanism
Glucuronidation metabolism — no oxidative (CYP) step, no active metabolites
Effect
Predictable clearance, unaffected by hepatic dysfunction or CYP interactions
Clinical applications
Preferred benzodiazepine in liver disease and in older adults (with diazepam and oxazepam — the 'LOT' agents safe in hepatic impairment).
Mechanism
Intermediate half-life (~12h); reliable IM absorption
Effect
Predictable duration; effective parenteral option
Clinical applications
IM/IV lorazepam is a standard parenteral benzodiazepine for acute agitation and seizures.
Mechanism
CNS depression
Effect
Sedation, cognitive effects, falls; respiratory depression with opioids
Clinical applications
Standard benzodiazepine cautions apply, especially in older adults.

Mechanism note: Lorazepam's glucuronidation metabolism — no CYP step, no active metabolites — makes it the predictable, interaction-light benzodiazepine for hepatic impairment and medically complex patients.

For status epilepticus, IV lorazepam 4 mg is first-line per current guidelines. For procedural anxiolysis, oral or sublingual lorazepam works.

No active metabolites — directly glucuronidated and excreted. Useful in hepatic impairment (vs. diazepam, which accumulates active metabolites). Predictable kinetics across populations.

One distinctive use that deserves mention: catatonia. Lorazepam at 1-2 mg IM or IV is both diagnostic and therapeutic for catatonia. The response can be dramatic — patients with profound psychomotor immobility who haven't spoken for days will begin moving and talking within 30 minutes. If lorazepam works, continue scheduled lorazepam every 6-8 hours while the underlying cause is addressed. If lorazepam fails, ECT is highly effective. Catatonia is treatable, and missing it has serious consequences; lorazepam challenge is the test.

Catatonia first-line: lorazepam often dramatically effective. Patient with profound psychomotor immobility may respond within hours. Diagnostic and therapeutic — both confirms diagnosis and treats.

Lorazepam has the qualities you want in an acute BZD: predictable, versatile, well-tolerated, accessible across hepatic conditions. For acute settings, it is the standard tool.

Prescribing reality
Cost
Generic: ~$10-25/month.
Generic status
Generic for decades.
Formulary typical
Tier 1 generic.
Access friction
Schedule IV scheduling. IM/IV forms universally available in acute settings.

Prescriber tip: Workhorse for acute psychiatric situations. For outpatient use, write small quantities, PDMP review at every prescription. Document indication and time-limited plan.

The anchor

Lorazepam is the workhorse benzodiazepine for acute psychiatric situations — multiple formulations, no active metabolites, predictable kinetics. First-line for catatonia and frequently used for acute agitation, alcohol withdrawal, status epilepticus.

Prove it

A patient with catatonic features (mutism, posturing, waxy flexibility) receives IV lorazepam 2 mg. Within 30 minutes the patient begins speaking and moving normally. What does this represent?

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