Stage 2: Antidepressants II — Atypical & Novel
Concept 3 of 8
R2.3

Trazodone

SARI — used primarily for sleep at sub-therapeutic-for-depression doses.

Trazodone today: primarily an off-label sleep aid at 25-100 mg. Original FDA approval was for depression at higher doses, but sedation makes daytime dosing impractical.

Trazodone is the antidepressant nobody uses as an antidepressant anymore. It is FDA-approved for major depression at doses of 150 to 400 milligrams, but at those doses the sedation is so severe that daytime functioning is usually impossible. What trazodone is used for, almost universally, is sleep — at doses of 25 to 100 milligrams at bedtime, off-label, and absolutely everywhere.

Drug card
Class
Serotonin antagonist and reuptake inhibitor (SARI)
Mechanism
Weak SERT inhibition + strong 5-HT2A antagonism; also alpha-1 antagonism (orthostasis) and H1 antagonism (sedation)
Typical dose
For depression 150-400 mg/day. For insomnia 25-100 mg at bedtime (off-label).
Half-life
~7 hours
FDA indications
MDD (FDA-approved). Off-label: insomnia (most common modern use).
Key adverse effects
Sedation, orthostatic hypotension, dry mouth, blurred vision, priapism (rare but emergent)

Most common modern use: off-label for insomnia at 25-100 mg. Priapism is rare but a true emergency — counsel male patients. Avoid for daytime depression dosing; sedation usually intolerable.

The mechanism explains the split personality. Trazodone is a "SARI" — serotonin antagonist and reuptake inhibitor. It weakly inhibits SERT, blocks 5-HT2A receptors strongly, antagonizes alpha-1 adrenergic receptors (causing orthostasis), and blocks H1 receptors (causing sedation). At low doses, the H1 and alpha-1 effects dominate — sleep, with some morning grogginess. At higher doses, the SERT inhibition contributes antidepressant effect, but the sedation persists.

At low doses, 5-HT2A + H1 + alpha-1 antagonism produces sleep without significant SERT effect. At higher doses, SERT inhibition contributes antidepressant effect.

For sleep, trazodone is a popular choice for several reasons. It is non-controlled — no DEA scheduling, no abuse liability, no requirement for refill restrictions. It does not produce tolerance the way benzodiazepines and z-drugs can. It can be used long-term without the dependence concerns of GABA modulators. It does not suppress REM sleep significantly. For a patient with chronic insomnia who needs a sleep aid but should not have a controlled substance, trazodone is a reasonable choice.

Mechanism in practice

Trazodone is a second textbook case of dose-dependent recruitment — a hypnotic at low dose, an antidepressant only at high dose, because different receptors engage at different exposures.

Mechanism
H1 antihistamine, 5-HT2A, and alpha-1 antagonism (dominant at low doses)
Effect
Sedation; sleep onset and maintenance
Clinical applications
At 25-100mg trazodone is a hypnotic — the dominant real-world use. No respiratory depression, no dependence, weight-neutral.
Mechanism
SERT blockade recruited at higher doses (150-300mg+)
Effect
Antidepressant effect appears only at the higher dose range
Clinical applications
The patient on trazodone 50mg for sleep is NOT being treated for depression — the antidepressant mechanism is not yet engaged.
Mechanism
Alpha-1 adrenergic antagonism
Effect
Orthostatic hypotension; priapism (rare)
Clinical applications
Fall risk in older adults; counsel men about priapism as a urologic emergency.
Mechanism
5-HT2A antagonism
Effect
No sexual dysfunction; sleep architecture preservation
Clinical applications
Useful as a sleep agent when SSRI-induced insomnia or sexual dysfunction is the issue.

Mechanism note: Trazodone's split personality by dose — hypnotic low, antidepressant high — is dose-dependent receptor recruitment in action. Know which mechanism the prescribed dose actually engages.

Side effects are mostly the H1 and alpha-1 effects. Morning grogginess is the most common complaint, especially with doses over 50 milligrams. Orthostatic hypotension is meaningful in older patients — dizziness on standing, fall risk. Dry mouth, headache, occasional nausea.

The signature serious adverse effect — rare but unforgettable — is priapism. Trazodone can produce sustained, painful erection from alpha-1 blockade in penile vasculature. Untreated for more than four hours, priapism can cause permanent erectile dysfunction. Counsel every male patient about this at the time of prescription: a prolonged painful erection is a urological emergency, not something to wait out. Document the counseling.

Priapism — a rare but emergent adverse effect from alpha-1 blockade in penile vasculature. Permanent erectile dysfunction possible if untreated >4 hours.

Dosing for sleep is simple. Start 25 to 50 milligrams at bedtime. Some patients need 100 milligrams. Above that, the daytime sedation usually becomes intolerable for sleep purposes. For antidepressant effect, doses are 150 to 400 milligrams divided — daytime use is hard.

Prescribing reality
Cost
Generic: ~$4-10/month. On most $4 lists.
Generic status
Generic since 1989. Universally available.
Formulary typical
Tier 1 generic. No PA.
Access friction
None for sleep doses. Higher doses for depression are rare.

Prescriber tip: Cheapest non-controlled sleep aid. Counsel every male patient about priapism — rare but emergent, and many patients hear it for the first time only after the prescription is filled.

Drug interactions are modest at sleep doses. The 5-HT2A blockade reduces concern for serotonin syndrome compared to pure serotonergic agents, but trazodone is still serotonergic enough to add to the burden if combined with MAOIs or other strongly serotonergic drugs.

When a patient asks for "something for sleep" that isn't addictive, trazodone is often the answer. When you offer it, mention the priapism risk explicitly to male patients, and start low.

The anchor

Trazodone is FDA-approved for depression but used primarily off-label as a non-controlled sleep aid at 25-100 mg. The 5-HT2A/H1/alpha-1 antagonism produces sleep without the dependence risks of benzodiazepines or z-drugs.

Prove it

A 30-year-old male patient with depression is starting trazodone 50 mg for sleep. What specific counseling is essential?

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